The truth is in the middle, and the middle is uncomfortable for people who want clean answers
Neuropathological studies have provided evidence of reduced GABAR levels in the cortex and hippocampus, aberrant GAD1 and GAD2 mRNA expression in the postmortem cortex and cerebellum, and the interneuron markers parvalbumin (PV) and somatostatin (SST) are downregulated
These can destabilise the peptide and may cause irritation
Add a comment This will be publicly visible
They also highlight that lab variability in testing AMH can generate disparate results, making it difficult to compare such findings
Where exactly is the patient centred, solid, qualitative, peer-reviewed research from back when this ridiculously illogical idea originated